Add a listing
    0
    Add a listing

    How Diuretics Cause Hyperuricemia

    There is a great need for new effective treatment for hyperuricemia. The general population is increasingly becoming aware of its negative consequences on health. Because more than half of all patients with gout have no known cause, and gout is often left as a silent or inherited disease, it is imperative to find a cure. The National Institute of Arthritis and Musculoskeletal Disease (NIAMS), part of the National Institutes of Health, has recently announced the formation of a new scientific group whose primary goal will be to search for gout causes.

    Although there currently are no approved medications to treat hyperuricemia as a disease, several medications have been used in the past to treat the underlying cause of gout. Generally diuretics were prescribed for the secondary hyperuricemia as a means of decreasing further loss of fluids, especially urine. Although diuretics were found to be safe and effective for most patients with gout, there was one category of patients that became unresponsive to diuretics such as patients with kidney disease. As a result, in those cases, the medical community began to look at other options for treating hyperuricemia.

    In recent years, scientists have learned that obesity and hypertension are often caused by decreased production of fibrin, a component of cell walls in the body. Although obesity and hypertension are often thought of as inevitable diseases, decreased fibrin levels can often be treated with gout drugs. In addition to treating the primary cause, gout drugs are sometimes beneficial in preventing future gout attacks. For example, if a person with gout suffers from sudden increases in blood pressure, a decrease in the concentration of uric acid may prevent or reduce the likelihood of developing future gout attacks.

    Some individuals respond well to diuretics while others do not. Also, there is a difference between acute and chronic hyperuricemia. For example, an individual who is experiencing symptoms of hyperuricemia might need to take diuretics for a few days or weeks to bring blood flow back to normal. On the flip side, an individual with an acute onset of hyperuricemia may already be taking diuretics and is therefore more likely to benefit from these medications. If you have an acute onset of hyperuricemia and are currently taking diuretics to bring your blood sugar levels back into a healthy range, talk to your doctor about changing your prescription.

    Generally, individuals with a family history of gout or high blood pressure are more likely to develop hyperuricemia or a similar condition. Other causes that can result in hyperuricemia include alcohol abuse, liver disease, and kidney disease. A urate crystal in the kidney can block blood flow and increase uric acid production. This increased production of uric acid can cause the formation of crystals in the vein walls that cause gout or high blood pressure. In order to avoid the development of one of these conditions, it’s important that you make sure to eat a healthy diet and get regular physical activity.

    Hyperuricemia has many potential causes and it’s important to know them in order to choose the best treatment for your specific case. Although BMI or body mass index is not considered a primary cause of hyperuricemia or any other heart disease, it is often a factor. Those who have a higher BMI than their normal range (which is about 30) are more likely to experience some of the other health conditions linked to obesity. There are two ways to monitor your health: by using a blood pressure gauge or with a time-varying blood pressure watch. While a good diet and exercise regimen are still essential for optimal health, you may want to consider using an EGBM to see how you’re progressing.

    More Gout Resources

    Comments

  • No comments yet.
    • Add a comment

    Leave a Reply ·